Research Highlights | Regional Centre for Biotechnology

The illustration depicts the RICTOR–UGCG axis as a control circuit that rewires cancer cell metabolism. Activated RICTOR disrupts epigenetic regulators to boost UGCG-driven GD3 ganglioside production, which in turn hyperactivates EGFR signalling. Together, this metabolic–epigenetic circuitry fuels sustained cancer cell proliferation

mTORC2 Driven Metabolic-Epigenetic Rewiring of Gangliosides Promotes Breast Cancer Progression

Photograph of two uprooted young plants placed on a dark background, shown with intact shoots and root systems for comparison.

A method to identify DNA sequences flanking retrotransposon insertion sites in chickpea

Schematic illustration of pancreatic cancer showing a regulatory loop driven by epigenetics. Epigenetics-driven differential transcription upregulates NFAT2, leading to transcriptional upregulation of the calcium channel Orai3 at the plasma membrane, while March8 promotes lysosomal degradation.

One Factor, Two Fates: How NFATc1 Controls Orai3 in Pancreatic Cancer

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